ATAR Notes: Forum
Uni Stuff => Universities - Victoria => University of Melbourne => Topic started by: Turtle on March 29, 2014, 10:39:07 am
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Does anyone understand slide 21 of lecture 12?
It makes absolutely no sense to me :o
Also, can someone please explain glycogen super-compensation to me if possible?
Thanks!!
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Not sure if you were still wondering about glycogen supercompensation but this was what I wrote
Non-‐exercised
legs:
Muscle
glycogen
levels
remained
relatively
constant
Exercised
legs:
Muscle
glycogen
levels
returned
to
resting
after
one
day
and
after
two-‐three
days
muscle
glycogen
levels
increased
(supercompensation)
^ That copied & pasted badly. I'm not sure how in-depth we have to go into for this, but looking at some stuff on the net:
"it is just the process of increasing anaerobic energy stores (glycogen) beyond the average capacity. "
"supercompensation is the post training period during which the trained function/parameter has a higher performance capacity than it did prior to the training period"
So I think it's like this:
Excercise muscles -> Post-exercise period ingest CHO -> Replenish glycogen stores initially and then have glycogen stores increase beyond the average level -> Increased glycogen storage means greater power output for future exercise. Just one of the many adaptations to exercise I think.
-
Not sure if you were still wondering about glycogen supercompensation but this was what I wrote
Non-‐exercised
legs:
Muscle
glycogen
levels
remained
relatively
constant
Exercised
legs:
Muscle
glycogen
levels
returned
to
resting
after
one
day
and
after
two-‐three
days
muscle
glycogen
levels
increased
(supercompensation)
^ That copied & pasted badly. I'm not sure how in-depth we have to go into for this, but looking at some stuff on the net:
"it is just the process of increasing anaerobic energy stores (glycogen) beyond the average capacity. "
"supercompensation is the post training period during which the trained function/parameter has a higher performance capacity than it did prior to the training period"
So I think it's like this:
Excercise muscles -> Post-exercise period ingest CHO -> Replenish glycogen stores initially and then have glycogen stores increase beyond the average level -> Increased glycogen storage means greater power output for future exercise. Just one of the many adaptations to exercise I think.
Thanks El, that was really helpful!! :)
I'm glad it wasn't on the mid semester test!
How did you find the MST? I thought it was moderate in terms of difficulty. It definitely wasn't easy. I think a lot of people would have been a bit complacent about it, and they would have found it hard. It had a lot of little detail questions in it.
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Thanks El, that was really helpful!! :)
I'm glad it wasn't on the mid semester test!
How did you find the MST? I thought it was moderate in terms of difficulty. It definitely wasn't easy. I think a lot of people would have been a bit complacent about it, and they would have found it hard. It had a lot of little detail questions in it.
Probably not anticipating a great result to be honest.
Exercise metabolism is not my friend.
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I thought GLUT4 transporters and PP1 were bound to intramuscular glycogen, and when you used up the glycogen stores this released GLUT4 and PP1 into the cell. During/after exercise GLUT4 is then able to translocate to the membrane and allow more glucose to enter the cell. The PP1 then phosphorylases glycogen synthase, making it active. So the increase in glucose translocation into the cell (from GLUT4) + increased glycogen synthase activity causes a super-compensatory effect and creates way too much glucose over the 2-3 days post exercise.
I might be wrong but it kind of makes sense...I think...
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way too much glycogen i mean!!
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I thought GLUT4 transporters and PP1 were bound to intramuscular glycogen, and when you used up the glycogen stores this released GLUT4 and PP1 into the cell. During/after exercise GLUT4 is then able to translocate to the membrane and allow more glucose to enter the cell. The PP1 then phosphorylases glycogen synthase, making it active. So the increase in glucose translocation into the cell (from GLUT4) + increased glycogen synthase activity causes a super-compensatory effect and creates way too much glucose over the 2-3 days post exercise.
I might be wrong but it kind of makes sense...I think...
Sure, those are the possible combined mechanisms as to how glycogen resynthesis may arise, but I was just trying to bring up the reasoning as to why it probably occurs :)