ATAR Notes: Forum
VCE Stuff => VCE Science => VCE Mathematics/Science/Technology => VCE Subjects + Help => VCE Biology => Topic started by: bar0029 on October 28, 2010, 03:37:08 pm
-
VNers this is our time to shine,
time to show the assessors what we really are made of
time to get those top 10% of the state scores
we are the ones that put all our effort in
we have achieved everything we have done so far
goodluck tomorrow
you've worked hard, we deserve to get the best out of tomorrow
just remember, while you're writing away tomorrow,
and you're thinking oh i wish i was the smartest person in the class..
well guess what you are!!!!!!!!!!!!
because phenotype = genotype x environment
and your environment has been biology all semester long *corny*
GOODLUCK! :smitten:
-
LOL :)
-
Someone see if they can grab a spare copy of the paper, I'm curious to see it :)
-
LOL Russ. All I know is that it's 25 pages long :P
Imagine if it were as long (in terms of content) as mid year!?
-
Would you expect that there would be a question about designing an experiment? What's the best way to approach questions like these?
-
LOL Russ. All I know is that it's 25 pages long :P
Imagine if it were as long (in terms of content) as mid year!?
I'm counting on you now :P
I don't think it'll be as laborious as the mid year, I think they'll put an emphasis on biological reasoning. But knowing them, it'll end up being 90% regurgitation of facts (I jinxed it).
If you have to design an experiment, just make sure you clearly state your hypothesis and how your design will test it. So if your hypothesis that a gene is in a certain, state that you'll be using restriction enzymes to check etc.
-
bio is easy you can lose like 10-15 marks and still get A+... i mean what other subject allows that?
-
That means it's harder, because there are less people getting high marks...
(ie renaissance or classics, where getting full marks on the exam + full marks on sacs and you can still get less than 50)
-
Really? Then how do they distinguish who gets 50 and who doesn't?
-
we go through designing an experiment in unit 3, while i suppose it's assumed knowledge it isn't really part of unit 4 so if they did it'd be cruel and if it were more than a 1 mark then...riots would result.
and
i have done too much bio revision, as bar said VNers time to shine, let's kill it. goodluck allz.
-
VNers this is our time to shine,
time to show the assessors what we really are made of
time to get those top 10% of the state scores
we are the ones that put all our effort in
we have achieved everything we have done so far
goodluck tomorrow
you've worked hard, we deserve to get the best out of tomorrow
just remember, while you're writing away tomorrow,
and you're thinking oh i wish i was the smartest person in the class..
well guess what you are!!!!!!!!!!!!
because phenotype = genotype x environment
and your environment has been biology all semester long *corny*
GOODLUCK! :smitten:
Thats touching... in a sciency kind of way
Really? Then how do they distinguish who gets 50 and who doesn't?
With hard questions
-
I meant for Classics: where 100% in SACs and exam doesn't guarantee a 50 :P
-
we go through designing an experiment in unit 3, while i suppose it's assumed knowledge it isn't really part of unit 4 so if they did it'd be cruel and if it were more than a 1 mark then...riots would result.
and
i have done too much bio revision, as bar said VNers time to shine, let's kill it. goodluck allz.
On a few VCAA exams there have been questions relating with experiments, worth 3 marks.
-
Really? Then how do they distinguish who gets 50 and who doesn't?
GAT marks apparently.
-
If the question states name the trait is codominant, would the alleles be labelled for example IB as opposed to B? Or does the definition of an allele fit that it would be named B?
-
LOL Russ. All I know is that it's 25 pages long :P
Imagine if it were as long (in terms of content) as mid year!?
How do you know it's going to be 25 pages long?
-
http://www.vcaa.vic.edu.au/vcaa/vce/exams/examcovers/Oct_Nov_2010/2010biol2-cov.pdf
Materials supplied
• Question and answer book of 25 pages.
• Answer sheet for multiple-choice questions.
-
you do Ito the B because this shows co-dominance :)
-
you do Ito the B because this shows co-dominance :)
Really? Co-dominance occurs when the contributions of both alleles are visible in the phenotype. In the ABO example, the IA and IB alleles are co-dominant in producing the AB blood group phenotype, in which both A- and B-type antigens are made. (Wikipedia).
-
Incomplete dominance is not in the study design actually :P
And lucky it is, because it gets confusing
Example: HbA and HbS alleles for the gene controlling haemoglobin production
Consider the genotype HbA HbS.
phenotypes "production of haemoglobin A" and "production of haemoglobin S" are co-dominant
phenotypes "sickle-cell anaemia" and "no sickle-cell anaemia" are incompletely dominant
The same genotype can control more than one phenotype, which may have different dominance relationships (which is why we refer to PHENOTYPES not genotypes as being dominant or recessive).
-
Incomplete dominance is not in the study design actually :P
And lucky it is, because it gets confusing
Example: HbA and HbS alleles for the gene controlling haemoglobin production
Consider the genotype HbA HbS.
phenotypes "production of haemoglobin A" and "production of haemoglobin S" are co-dominant
phenotypes "sickle-cell anaemia" and "no sickle-cell anaemia" are incompletely dominant
The same genotype can control more than one phenotype, which may have different dominance relationships (which is why we refer to PHENOTYPES not genotypes as being dominant or recessive).
It was on the 2006 VCAA paper question 10 multiple choice. But from the other answers you could deduce it had to be that one.
-
Really? the phenotypes are dominant or recessive? isn't it the genotypes, because the trait will be expressed as the phenotype depending on which one is dominant??
-
Incomplete dominance is not in the study design actually :P
And lucky it is, because it gets confusing
Example: HbA and HbS alleles for the gene controlling haemoglobin production
Consider the genotype HbA HbS.
phenotypes "production of haemoglobin A" and "production of haemoglobin S" are co-dominant
phenotypes "sickle-cell anaemia" and "no sickle-cell anaemia" are incompletely dominant
The same genotype can control more than one phenotype, which may have different dominance relationships (which is why we refer to PHENOTYPES not genotypes as being dominant or recessive).
Is that because the environment plays a role?
-
Incomplete dominance won't be used because it's really controversial over what is incomplete and what isn't.
ie. a usual example is a pink flower from a red and white flower -> used to be called incomplete dominance
but it's actually codominance as both red and white is expressed making it look pink to us.
-
That's the thing: it depends on the actual phenotype you are talking about. It's really a debatable topic - let's use that example about the white-red-pink flowers:
'production of white pigment' and 'production of red pigment' are co-dominant
BUT
'white flower' and 'red flower' are incompletely dominant
And yes, phenotypes NOT genotypes are dominant or recessive. Never use "dominant allele" - got that from Douchy
Douchy's awesome!
-
Yer I love douchy too :D sooo... The trait is dominant or recessive, but not the actual phenotype itself? this is confusing me, since the phenotype is genotype + environment therefore a "phenotype" couldn't be recessive or dominant, could it?
-
A phenotype can be dominant. Usually what distinguishes recessive from dominant is that dominant produces (more) proteins, making it dominant. Recessive phenotypes may not produce the protein.
A phenotype involves the biochemical, physiological and physical expression of the genotype. An expression (or not) of a protein therefore involves the phenotype.
-
You may be right but..I am pretty sure that no matter what the environment throws at you, your genetically determined traits will not change or swap from one to the other. You get me?
-
Your environment can alter your genetic traits, it's the new big area of genetics research
e, it doesn't change the base sequence of your genome, but it can still have big effects
-
A read on wikipedia once that lemarcks theories are getting a little proof (but not exactly in the form he said them)
-
What I am saying though IS you would not say that your phenotype is dominant or recessive, im not saying that your genotype is either but your traits are dominant or recessive. For example you are heterzygous. Your environment will NOT change your genetically determined phenotypic trait from dominant to recessive Therefore you cannot claim your phenotype is dominant or recessive. Agree?
-
GOODLUCK EVERYONE LAST TIME I SEE BIO IN MY ENTIRE LIFE TOMORROW!!!!!!!!! :]
-
yeah. his reasons were wrong, because there was no way he could have known about what we're discovering now but his theory is actually being rekindled.