Pathology question thread

[BelVP]

Some of my answers which I could remember...
I answered "myofibroblasts" in the question regarding granulation tissue/the motorcycle as it seemed to me more like second intention healing.
Hyperplasia was definitely the cause of the monkey bar callous, I should know having experience them.
Primary chronic inflammation is chronic inflammation which does not progress directly from acute inflammation hence it could not have been prolonged acute inflammation.

[nubs]

Ah I only just saw this thread, I know I'm a week late butttt I thought that the answer to the second motorcycle accident was fibroblasts and endothelial cells. It made sense for endothelial cells to be present, I thought, because within the first few days we have a load of new blood vessels being formed at the site of injury, and blood vessels are made up of endothelial cells.

Also, I didn't choose the myofibroblast + eosinophil one because I think eosinophils are usually only present during parasitic infection

Can someone explain to me why the formation of transduate is non-inflammatory?
I don't get it...because it is formed during the inflammatory process? 

I know that the transduate is formed due to an increase in hydrostatic pressure in the blood vessels, which is due to vasodilation. The increase in hydrostatic pressure causes the formation of a transduate in the extravascular space.

Is it trying to say that a transduate can also be formed during non-inflammatory processes too? ie. if the volume of blood flow increased for other reasons other than inflammation

I know that this is way late, but from how I interpreted it, a transudate is usually associated with non-inflammatory conditions, and forms due to increased hydrostatic pressure or decreased oncotic pressure in the blood vessels. So inflammatory conditions are not required for its formation, other things can bring it about.

From what I gathered from the CAL practical (and not the lectures) is that a transudate initially forms during the inflammatory response, and then when vascular permeability increases, it turns into an exudate as more and more proteins are able to pass through along with the fluid.

[Turtle]

Ah I only just saw this thread, I know I'm a week late butttt I thought that the answer to the second motorcycle accident was fibroblasts and endothelial cells. It made sense for endothelial cells to be present, I thought, because within the first few days we have a load of new blood vessels being formed at the site of injury, and blood vessels are made up of endothelial cells.

Also, I didn't choose the myofibroblast + eosinophil one because I think eosinophils are usually only present during parasitic infection


I know that this is way late, but from how I interpreted it, a transudate is usually associated with non-inflammatory conditions, and forms due to increased hydrostatic pressure or decreased oncotic pressure in the blood vessels. So inflammatory conditions are not required for its formation, other things can bring it about.

From what I gathered from the CAL practical (and not the lectures) is that a transudate initially forms during the inflammatory response, and then when vascular permeability increases, it turns into an exudate as more and more proteins are able to pass through along with the fluid.

Thanks Nubs!! I thought the same thing about the transduate, but you confirmed it for me

[Turtle]

Pathology marks are up!!!! 
I got 29/30, which I am really happy with!!

[BelVP]

Yay. I got 25/30, not too shabby 

[pink0829]

Got 27!! So happy!!

[Sinner]

Took them long enough. 27, my best so far!

[nubs]

28

[hobbitle]

Hey all you guys doing Path majors...
Are you all planning to do an MD afterwards?

[Turtle]

Hey all you guys doing Path majors...
Are you all planning to do an MD afterwards?

I'm hoping to do medicine after Science, except I'm undecided between a pathology, or human structure and function major.

[Starlight]

I'm hoping to do medicine after Science, except I'm undecided between a pathology, or human structure and function major.

Haha I thought you were set on HSF turtle, i'm sure you'll do great in either

[Turtle]

Haha I thought you were set on HSF turtle, i'm sure you'll do great in either

I'm 90% sure HSF, 10% sure Pathology.
At the moment, I'm still thinking HSF though!!
Thanks El, I'm sure you will too

[Turtle]

How did everyone find the 2nd MST? 
I found it about the same as the first, in terms of difficulty.
Can someone explain to me...wtf is "air flow" as a mechanical barrier to infection?
I chose that one as the incorrect one.
Did we even study that....?

[nubs]

How did everyone find the 2nd MST? 
I found it about the same as the first, in terms of difficulty.
Can someone explain to me...wtf is "air flow" as a mechanical barrier to infection?
I chose that one as the incorrect one.
Did we even study that....?

I thought it was okay overall, but still a fair bit harder than the first one
And yeah the airflow stuff is straight off the lecture slides. She said that breathing in and breathing out makes it harder for pathogens to adhere to the cells, cause of all the air flowing.

I put the answer as normal flora but I wasn't 100% sure at the time :/

[nubs]

What about the question that went something like, 'A pathogen can cause a disease in all of the following ways except:
Secreting endotoxins
Secreting exotoxins
Making contact with the cell
And there was one other one

I chose making contact with the cell, since there needs to be more than just contact. Like there needs to be adhesion and stuff or something, but I really wasn't sure. What did you guys put down?