Yeah sorry my bad! Had a lot of questions asking about it... I simply thought that the synthesised product (I forgot its name now) attaches to the allosteric region of the ATCase --> active site shape changes --> cannot properly bind to A and B substrate, production rate of D decreases as a result.
^ in simplified terms, of course.
Yeah I wrote that the high production of CTP inhibits the rate of the enzyme ATCase in which the production of D is regulated in a decrease - thus an equilibrium. I didn't put much detailed into it however realising it was worth 3 marks now I wish I did haha
What did everyone write for question 3 in short answer? And was question 10 part a and b both monohybrid crosses? (if not then I am so dead
)
For 3a. I wrote about the binding to a plasma membrane receptor because of the hydrophilic property of the hormone. This initiates secondary messengers in which signal transduction takes place - cascade of events/chemical reactions. This then stimulates glycogenolysis where glycogen is converted to glucose. Glucose then leaves the liver going into the blood.
3b. A net movement of glucose molecules from a higher glucose concentration -inside the cell- to a lower glucose concentration -outside the cell-. The high concentration facilitates the movement outside the cell hence glucose molecules released in the blood. (I forgot to mention that it needed protein carriers and that it was passive - not requiring energy. Ah well hope I get the marks)
3c. Mitochondria the site of aerobic respiration - energy production. (32-34 ATP). I then went on to explain cellular respiration and how the mitochondria is needed for cells that require a constant supply of oxygen.
For 10 you might want to check Scooby's suggested answers! -
2014 VCE Biology Exam Solutions by Scooby